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Rhoden EL, Morgentaler A. Risks of testosterone-replacement remedy and recommendations for monitoring. Testosterone substitute in hypogonadal men with angina improves ischaemic threshold and high quality of life. Ajayi AAL, Mathur R, Halushka PV. Testosterone increase human platelet thromboxane A2 receptor density and aggregation responses.
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- The affected person underwent profitable percutaneous intervention of the right coronary artery.
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- Anabolic androgenic steroid use has increased over the previous decade, despite its potential for antagonistic results.
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Anabolic Steroids, Acute Myocardial Infarction And Polycythemia: A Case Report And Evaluation Of The Literature
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We report a case of an acute ST-segment elevation myocardial infarction in a patient with traditional cardiac threat elements utilizing supraphysiologic doses of supplemental, intramuscular testosterone. In addition, this patient additionally had polycythemia, doubtless secondary to high-dose testosterone. The affected person underwent profitable percutaneous intervention of the proper coronary artery. Physicians and sufferers should pay attention to the clinical penalties of anabolic steroid abuse. Development of an atheromatous plaque perpetuates endothelial dysfunction and promotes platelet aggregation and intracoronary thrombus formation (Ajayi et al 1995; Nieminen et al 1996).
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Moreover, a part of endothelial dysfunction has been proposed, which may contribute to irregular vessel reactivity. However, testosterone in physiologic doses could even be useful in patients with angina , have higher ischemic thresholds and improved high quality of life . Short-term intracoronary administration of testosterone induces coronary artery dilation and will increase coronary blood flow in males with established coronary artery disease . Therefore, an interaction of endogenous components of the patient’s danger profile in addition to the testosterone dosing may be issues of whether or not antagonistic events occur.
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